السبت، 22 يناير 2011

What makes you throw up after you run really hard?

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Best Answer - Chosen by Asker
 
When you run at a maximal effort, your body is going to send blood to those areas that are doing the most work. Your heart is pumping lots of blood, and oxygen, to your legs and arms. It is going to take away from nonessential functions such as digestion. If you push hard enough, it will actually take away from your brain -- ever feel lightheaded? Also if you push hard enough, you are pushing your body to a point of extreme imbalance, and then when you end your race, your body is trying to get back to normal. It is trying to clean up the lactic acid, trying to bring back the calcium into the cells, get oxygen everywhere that your stomach becomes the weakened area and you throw up.

Let me put another way. When you are racing you are bracing your stomach muscles very tightly which is putting a tremendous amount of pressure on your stomach. When you stop, you instantaneously relax those muscles. Now the stomach is going to try to go back to its original shape and guess what, it does too fast and you don't have the muscle control to keep your food down.

If you train correctly, you will never throw up. You should be pushed, but throwing up if typically due to improper training methods and you will not get as much from the workout if you throw up.

Good Luck!
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WIKI said
During power exercises such as sprinting, when the rate of demand for energy is high, lactate is produced faster than the ability of the tissues to remove it, so lactate concentration begins to rise. This is a beneficial process, since the regeneration of NAD+ ensures that energy production is maintained and exercise can continue. The increased lactate produced can be removed in a number of ways, including:
  • Oxidation to pyruvate by well-oxygenated muscle cells, which is then directly used to fuel the Krebs cycle
  • Conversion to glucose via gluconeogenesis in the liver and release back into the circulation; see the Cori cycle.
Contrary to popular belief, this increased concentration of lactate does not directly cause acidosis, nor is it responsible for delayed onset muscle soreness.[7] This is because lactate itself is not capable of releasing a proton,[7] and, second, the acidic form of lactate, lactic acid, "is not produced in muscle".[8] Analysis of the glycolytic pathway in humans indicates that there are not enough hydrogen ions present in the glycolytic intermediates to produce lactic or any other acid.
The acidosis that is associated with increases in lactate concentration during heavy exercise arises from a separate reaction. When ATP is hydrolysed, a hydrogen ion is released. ATP-derived hydrogen ions are responsible primarily for the decrease in pH. During intense exercise, aerobic metabolism cannot produce ATP quickly enough to supply the demands of the muscle. As a result, anaerobic metabolism becomes the dominant energy-producing pathway, as it can form ATP at high rates. Due to the large amounts of ATP being produced and hydrolysed in a short period of time, the buffering systems of the tissues are overcome, causing pH to fall and creating a state of acidosis, a natural process that facilitates the easier dissociation of oxyhaemoglobin and allows easier transfer of oxygen from the blood.[9]
The effect of lactate on acidosis has been the topic of many recent conferences in the field of exercise physiology. Robergs et al. have accurately chased the proton movement that occurs during glycolysis. However, in doing so, they have suggested that [H+] is an independent variable that determines its own concentration. A recent review by Lindinger et al.[8] has been written to rebut the stoichiometric approach used by Robergs et al.[7] In using this stoichiometric process, Robergs et al. have ignored the causative factors (independent variables) of the concentration of hydrogen ions (denoted [H+]). These factors are strong ion difference [SID], PCO2, and weak acid buffers. Lactate is a strong anion, and causes a reduction in [SID], which causes an increase in [H+] to maintain electroneutrality. PCO2 also causes an increase in [H+]. During exercise, the intramuscular lactate concentration and PCO2 increase, causing an increase in [H+], and, thus, a decrease in pH. (See Le Chatelier's principle)

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